International Journal of Academic Medicine

: 2017  |  Volume : 3  |  Issue : 3  |  Page : 72--76

Polycystic kidney disease

Stanislaw P Stawicki1, Gary Lombardo2 
1 OPUS 12 Foundation, Bethlehem Chapter, PA, USA
2 OPUS 12 Foundation, Bethlehem Chapter; Department of Surgery, Division of Traumatology and Surgical Critical Care, University of Pennsylvania School of Medicine, Philadelphia, PA, USA

Correspondence Address:
Stanislaw P Stawicki
Department of Research and Innovation, St. Luke's University Health Network, 801 Ostrum Street, Bethlehem, PA 18015

Key points: (a) Polycystic kidney disease (PKD) is one of the most common genetic disorders; (b) There are two main types of PKD– autosomal dominant (most common) and autosomal recessive; (c) Diagnosis of PKD relies on a combination of clinical history, family history, imaging studies (computed tomography, ultrasound) and clinical findings (hematuria, hypertension, proteinuria); (d) Extrarenal manifestations of PKD include hepatic cysts, pancreatic and intestinal cysts, colonic diverticula, inguinal and abdominal wall hernias, valvular heart disorders, and cerebral aneurysms; (e) Treatment is centered on ameliorating symptoms and providing supportive care– therapy consists of adequate analgesia for pain symptoms, antibiotics for urinary tract infections, antihypertensive medications, renal supportive therapy; (f) Surgical indications include surgical decompression of large cysts and renal transplantation for end-stage renal failure. The following core competencies are addressed in this article: Medical knowledge and patient care. Republished with permission from: Stawicki SP, Lombardo G. Polycystic kidney disease. OPUS 12 Scientist 2008;2(1):17-20.

How to cite this article:
Stawicki SP, Lombardo G. Polycystic kidney disease.Int J Acad Med 2017;3:72-76

How to cite this URL:
Stawicki SP, Lombardo G. Polycystic kidney disease. Int J Acad Med [serial online] 2017 [cited 2022 Aug 15 ];3:72-76
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